[1] |
韦志明, 方汉军, 李中峰, 等. 痛风性膝关节炎的手术治疗进展[J/OL]. 中华关节外科杂志( 电子版), 2022, 16( 3 ): 343-347.
|
[2] |
Dalbeth N, Gosling AL, Gaffo A, et al. Gout[J]. Lancet, 2021, 397( 10287 ): 1843-1855.
|
[3] |
方宁远,吕力为,吕晓希,等.中国高尿酸血症相关疾病诊疗多学科专家共识(2023年版)[J].中国实用内科杂志, 2023, 43( 06 ):461-480.
|
[4] |
Cabău G, Crișan TO, Klück V, et al. Urate-induced immune programming: consequences for gouty arthritis and hyperuricemia [J].Immunol Rev, 2020, 294( 1 ): 92-105.
|
[5] |
Wang C, Ma C, Gong L, et al. Macrophage polarization and its role in liver disease[J/OL]. Front Immunol, 2021, 12: 803037.DOI:10.3389/fimmu.2021.803037.
|
[6] |
Bousoik E, Qadri M, Elsaid KA. CD44 receptor mediates urate crystal phagocytosis by macrophages and regulates inflammation in A murine peritoneal model of acute gout[J/OL]. Sci Rep, 2020, 10( 1 ): 5748. DOI:10.1038/s41598-020-62727-z.
|
[7] |
Michalick L,Kuebler WM. TRPV4-a missing link between mechanosensation and immunity[J/OL]. Front Immunol, 2020, 11:413. DOI:10.3389/fimmu.2020.00413.
|
[8] |
Lan Z, Chen L, Feng J, et al. Mechanosensitive TRPV4 is required for crystal-induced inflammation [J]. Ann Rheum Dis, 2021, 80( 12 ): 1604-1614.
|
[9] |
Silva CR, Saraiva AL, Rossato MF, et al. What do we know about toll-like receptors involvement in gout arthritis?[J]. Endocr Metab Immune Disord Drug Targets, 2023, 23( 4 ): 446-457.
|
[10] |
Zhao J, WeiK, JiangP, etal. Inflammatory response to regulated cell death in gout and its functional implications[J/OL]. Front Immunol,2022, 13: 888306. DOI:10.3389/fimmu.2022.888306.
|
[11] |
CoboI, Cheng A, Murillo-Saich J, et al. Monosodium urate crystals regulate a unique JNK-dependent macrophage metabolic and inflammatory response[J/OL]. Cell Rep, 2022, 38( 10 ): 110489.DOI:10.1016/j.celrep.2022.110489.
|
[12] |
Liu L, Zhu L, LiuM, etal. Recent insights into the role of macrophages in acute gout[J/OL]. Front Immunol, 2022,13:955806.DOI:10.3389/fimmu.2022.955806.
|
[13] |
Zhao L, Ye W, Zhu Y, et al. Distinct macrophage polarization in acute and chronic gout[J]. Lab Invest, 2022, 102( 10 ): 1054-1063.
|
[14] |
Klück V, Cabău G, Mies L, et al. TGF-β is elevated in hyperuricemic individuals and mediates urate-induced hyperinflammatory phenotype in human mononuclear cells[J/OL]. Arthritis Res Ther,2023, 25( 1 ): 30. DOI:10.1186/s13075-023-03001-1.
|
[15] |
Silvestre-Roig C, Fridlender ZG, Glogauer M, et al. Neutrophil diversity in health and disease[J]. Trends Immunol, 2019, 40( 7 ):565-583.
|
[16] |
Papayannopoulos V. Neutrophil extracellular traps in immunity and disease[J]. Nat Rev Immunol, 2018, 18( 2 ): 134-147.
|
[17] |
Huang J, Hong W, Wan M, et al. Molecular mechanisms and therapeutic target of NETosis in diseases[J/OL]. Med Comm, 2022,3( 3 ): e162. DOI:10.1002/mco2.162.
|
[18] |
Tang H, Tan C, Cao X, et al. NFIL3 facilitates neutrophil autophagy,neutrophil extracellular trap formation and inflammation during gout via REDD1-dependent mTOR inactivation[J/OL]. Front Med,2021, 8: 692781. DOI:10.3389/fmed. 2021.692781.
|
[19] |
Tan H, Li Z, Zhang S, et al. Novel perception of neutrophil extracellular traps in gouty inflammation[J/OL]. Int Immunopharmacol, 2023,115: 109642.DOI:10.1016/j.intimp.2022.109642.
|
[20] |
Tatsiy O, Mayer TZ, de Carvalho Oliveira V, et al. Cytokine production and NET formation by monosodium urate-activated human neutrophils involves early and late events, and requires upstream TAK1 and syk[J/OL]. Front Immunol, 2019, 10: 2996.DOI:10.3389/fimmu.2019.02996.
|
[21] |
Yin C, Liu B, Li Y, et al. IL-33/ST2 induces neutrophil-dependent reactive oxygen species production and mediates gout pain[J].Theranostics, 2020, 10( 26 ): 12189-12203.
|
[22] |
Reber LL, Gaudenzio N, Starkl P, et al. Neutrophils are not required for resolution of acute gouty arthritis in mice[J]. Nat Med, 2016, 22( 12 ): 1382-1384.
|
[23] |
Garcia-Gonzalez E, Gamberucci A, LucheriniOM, et al. Neutrophil extracellular traps release in gout and pseudogout depends on the number of crystals regardless of leukocyte count[J]. Rheumatology,2021, 60( 10 ): 4920-4928.
|
[24] |
Cumpelik A, Ankli B,Zecher D, et al. Neutrophil microvesicles resolve gout by inhibiting C5a-mediated priming of the inflammasome[J]. Ann Rheum Dis, 2016, 75( 6 ): 1236-1245.
|
[25] |
Sharabi A,Tsokos GC. T cell metabolism: new insights in systemic lupus erythematosus pathogenesis and therapy[J]. Nat Rev Rheumatol, 2020, 16( 2 ): 100-112.
|
[26] |
Wang B, Chen S, Qian H, et al. Role of T cells in the pathogenesis and treatment of gout[J/OL]. IntImmunopharmacol, 2020,88:106877. DOI:10.1016/j.intimp.2020.106877.
|
[27] |
Zhao LJ, Wang H, Gao HY, et al. Increase in different peripheral effector T subsets in acute and chronic gout[J/OL]. TransplImmunol,2023, 76:101763. DOI:10.1016/j.trim.2022.101763.
|
[28] |
Luo G, Yi T, Zhang G, et al. Increased circulating Th22 cells in patients with acute gouty arthritis: a CONSORT-compliant article[J/OL]. Medicine, 2017, 96( 42 ): e8329. DOI:10.1097/MD.0000000000008329.
|
[29] |
Liu X, Li Y, Li Z, et al. A novel IgG1 monoclonal antibody against xanthine oxidase alleviates inflammation induced by potassium oxonate in mice[J]. Int J Biol Macromol, 2018, 112: 537-547.
|
[30] |
Wang H, Xie L, Song X, et al. Purine-induced IFN-γ promotes uric acid production by upregulating xanthine oxidoreductase expression[J/OL]. Front Immunol, 2022, 13:773001. DOI:10.3389/fimmu.2022.773001.
|
[31] |
Dai XJ, Tao JH, Fang X, et al. Changes of treg/Th17 ratio in spleen of acute gouty arthritis rat induced by MSU crystals[J].Inflammation, 2018, 41( 5 ): 1955-1964.
|
[32] |
Raucci F, Iqbal AJ, Saviano A, et al. IL-17A neutralizing antibody regulates monosodium urate crystal-induced gouty inflammation[J/OL]. Pharmacol Res, 2019, 147: 104351. DOI:10.1016/j.phrs.2019.104351.
|
[33] |
Saviano A, Raucci F, Casillo GM, et al. Anti-inflammatory and immunomodulatory activity of Mangiferaindica L. reveals the modulation of COX-2/mPGES-1 axis and Th17/Tregratio[J/OL]. Pharmacol Res, 2022, 182:106283. DOI:10.1016/j.phrs.2022.106283.
|
[34] |
Migliorini P, Italiani P, Pratesi F, et al. The IL-1 family cytokines and receptors in autoimmune diseases[J/OL]. Autoimmun Rev,2020, 19( 9 ): 102617. DOI:10.1016/j.autrev. 2020.102617.
|
[35] |
Dinarello CA. Overview of the IL-1 family in innate inflammation and acquired immunity[J]. Immunol Rev, 2018, 281( 1 ): 8-27.
|
[36] |
Choe JY, Jung HY, Park KY, et al. Enhanced p62 expression through impaired proteasomal degradation is involved in caspase-1 activation in monosodium urate crystal-induced interleukin-1β expression[J]. Rheumatology ( Oxford ), 2014, 53( 6 ): 1043-1053.
|
[37] |
Heilig R, Dick MS, Sborgi L, et al. The Gasdermin-D pore acts as a conduit for IL-1β secretion in mice[J]. Eur J Immunol, 2018, 48 ( 4 ):584-592.
|
[38] |
Galvão I, Dias ACF, Tavares LD, et al. Macrophage migration inhibitory factor drives neutrophil accumulation by facilitating IL-1β production in a murine model of acute gout[J]. J Leukoc Biol, 2016,99( 6 ): 1035-1043.
|
[39] |
Gross O, Yazdi AS, Thomas CJ, et al. Inflammasome activators induce interleukin-1α secretion via distinct pathways with differential requirement for the protease function of caspase-1[J].Immunity, 2012, 36( 3 ): 388-400.
|
[40] |
Rider P, Carmi Y, Guttman O, et al. IL-1α and IL-1β recruit different myeloid cells and promote different stages of sterile inflammation[J]. J Immunol, 2011, 187( 9 ): 4835-4843.
|
[41] |
Fattori V, Staurengo-Ferrari L, Zaninelli TH, et al. IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis[J]. Inflamm Res, 2020, 69( 12 ): 1271-1282.
|
[42] |
Shang K, Wei Y, Su Q, et al. IL-33 ameliorates the development of MSU-induced inflammation through expanding MDSCs-like cells[J/OL]. Front Endocrinol, 2019, 10: 36. DOI:10.3389/fendo.2019.00036.
|
[43] |
Duan L, Huang Y, Su Q, et al. Potential of IL-33 for preventing the kidney injury via regulating the lipid metabolism in gout patients[J]. J Diabetes Res, 2016, 2016: 1028401.DOI:10.1155/2016/1028401.
|
[44] |
Liu L, Xue Y, Zhu Y, et al. Interleukin 37 limits monosodium urate crystal-induced innate immune responses in human and murine models of gout[J]. Arthritis Res Ther, 2016, 18( 1 ): 268.DOI:10.1186/s13075-016-1167-y.
|
[45] |
Ding L, Li H, Sun B, et al. Elevated interleukin-37 associated with tophus and pro-inflammatory mediators in Chinese gout patients[J/OL]. Cytokine, 2021, 141: 155468. DOI:10.1016/j.cyto.2021.155468.
|
[46] |
Zeng M, Dang W, Chen B, et al. IL-37 inhibits the production of pro-inflammatory cytokines in MSU crystal-induced inflammatory response[J].Clin Rheumatol, 2016, 35( 9 ): 2251-2258.
|
[47] |
KlückV, van DeurenRC, Cavalli G, et al. Rare genetic variants in interleukin-37 link this anti-inflammatory cytokine to the pathogenesis and treatment of gout[J]. Ann Rheum Dis, 2020, 79( 4 ): 536-544.
|
[48] |
Zhao L, ZhaoT, Yang X, et al. IL-37 blocks gouty inflammation by shaping macrophages into a non-inflammatory phagocytic phenotype[J]. Rheumatology, 2022, 61( 9 ): 3841-3853.
|
[49] |
杨智坚, 李奇. 白细胞介素-37与骨关节炎性疾病的研究进展[J/CD]. 中华关节外科杂志( 电子版 ), 2019, 13( 2 ): 200-205.
|